Repair & regenerative
Where tissue-repair biology meets the most-hyped corner of the peptide world — graded honestly.
thymosin β4 · GHK-Cu · BPC-157 · TB-500
Overview
Tissue repair is coordinated by peptide signals that regulate the cytoskeleton, angiogenesis, and inflammation. Thymosin β4 sequesters actin and supports wound healing; the copper-binding tripeptide GHK influences skin remodeling.
This family is also where the popular 'research peptides' cluster — compounds like BPC-157 and TB-500 that are widely discussed but, in most cases, supported predominantly by preclinical (animal) data. Each entry carries an explicit evidence badge so the biology stays separated from the marketing.
Key signals
Endogenous actin-sequestering peptide; wound healing and angiogenesis.
Endogenous copper-binding tripeptide; skin remodeling and repair signaling.
Synthetic peptide with broad preclinical repair claims; limited human data.
Synthetic thymosin β4 fragment marketed for recovery; preclinical evidence.
Reference notes
- Several entries here are synthetic 'research peptides' with limited or preclinical human evidence — read the evidence badge on each.
- Thymosin β4 (endogenous) and its fragment TB-500 are frequently conflated in the community; they are not the same thing.
- Honest evidence grading is the point: real biology, clearly separated from claims.