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PeptideHormone

Maridebart cafraglutide vs Glucagon-like peptide-1

GLP-1 agonism paired with GIP receptor antagonism — the opposite GIP direction to tirzepatide. The incretin that anchors the modern metabolic toolkit. Side by side: type, evidence tier, receptor, molecular weight, and half-life — the same fields as each monograph, rearranged so the engineering difference is visible.

Maridebart cafraglutide
MariTide
Glucagon-like peptide-1
GLP-1
Also known as
TypeAnalogEndogenous
EvidenceInvestigationalEstablished
FamilyIncretins & metabolicIncretins & metabolic
ClassGLP-1 agonist–GIP-antagonist peptide–antibody conjugateProglucagon-derived peptide (~30–31 aa)
ReceptorGLP-1 receptor (agonist) + GIP receptor (antagonist)GLP-1 receptor (GLP-1R), a class B GPCR
Molecular weight150,000 Da~3,297.7 Da
Half-life
~21 days (monthly dosing)
Model dosing
~1–2 min (native; DPP-4 cleaved)
Model dosing
Based onGlucagon-like peptide-1Native hormone

Half-life bars are on a logarithmic scale across the molecules shown. Reference values for the native or representative form — educational only, not medical or dosing advice.

Common questions

What is the difference between Maridebart cafraglutide and Glucagon-like peptide-1?
Maridebart cafraglutide is an engineered analog based on Glucagon-like peptide-1; Glucagon-like peptide-1 is an endogenous hormone. Maridebart cafraglutide signals at GLP-1 receptor (agonist) + GIP receptor (antagonist); Glucagon-like peptide-1 signals at GLP-1 receptor (GLP-1R), a class B GPCR.
How do the half-lives of MariTide and GLP-1 compare?
The reported circulating half-life of Maridebart cafraglutide is ~21 days (monthly dosing); Glucagon-like peptide-1 is ~1–2 min (native; DPP-4 cleaved). These are reference values for the native or representative form — educational only, not dosing advice.

Prefer the other order? Glucagon-like peptide-1 vs Maridebart cafraglutide. Or open the interactive comparison tool to add more molecules.